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This year’s American Association for Cancer Research (AACR) annual meeting took place on 1–5 April in Washington, DC, USA. The program committee Chair was Kornelia Polyak, MD, PhD, from the Dana-Farber Cancer Institute, Boston, Massachusetts.
On Sunday 2nd April, the “RAOS02 - Immunotherapy of Lymphoid Malignancies” session was introduced by its Chair, Margaret A. Shipp, MD, also from the Dana-Farber Cancer Institute, who gave a talk on immune evasion in Classical Hodgkin Lymphoma (cHL).
The talk began by discussing Reed-Sternberg (RS) cells, which are crippled CD30+ germinal center B-cells that display genomic instability. They are found in primary cHLs with an extensive inflammatory/immune cell infiltrate.
High throughput genomic assays are limited by the unique composition of cHLs. The precise incidence, nature, and prognostic impact of PD-L1/PD-L2 genetic alterations in cHL currently are unclear. The majority of newly diagnosed cHL patients display copy number alterations in PD-L1 and PD-L2, mostly copy gains and amplifications. High level alterations (amplification) can be predictive of outcome – negative standard for therapy. Therefore, does a genetic basis exist for sensitivity to PD-1 blockade?
Results of the CA209-205 registration trial indicated that high levels of 9p24.1 alterations and PD-L1 expression are associated with increased sensitivity to PD-1 blockade (Younes et al. Lancet Oncol. 2016).
Margaret Shipp also discussed data presented by Zinzani et al. at ASH 2016 of PD-1 blockade in R/R Mediastinal Large B-Cell Lymphoma (MLBCL). 9p24.1 amplification and PD-1L overexpression has been reported in some MLBCL patients (Green et al. Blood. 2010).
The talk then focused on “immune privilege” Lymphomas:
Margaret Shipp outlined findings reported by Nayak et al. during ASH 2016, exploring the use of nivolumab in Primary Central Nervous System Lymphoma (PCNSL) and Primary Testicular Lymphoma (PTL):
Following this the ways in which PD-1L overexpression occurs in lymphoid malignancies was discussed:
Margaret Shipp discussed findings published by Roemer et al. in Cancer Immunology Research in 2016, exploring the relationship between expression of β2M, MHC class I, and MHC class II, and 9p24.1 aberrations.
The talk was concluded by stating that this lack of β2M/MHC class I expression in RS cells in cHL has caused speculation regarding alternate mechanisms of PD-1 blockade in cHL.
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